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  • Writer's pictureIqraa Khaan

Understanding Actinic Keratosis – the Basics

Actinic keratosis (AK) is a common skin condition that arises due to long-term sun exposure. It is characterized by the development of rough, scaly patches on the skin. While actinic keratosis itself is not cancerous, it is considered a precancerous condition as it has the potential to progress into squamous cell carcinoma, a type of skin cancer. Understanding the basics of Actinic keratosis Treatment In Dubai can help individuals recognize its symptoms, seek appropriate treatment, and take preventive measures to protect their skin.

Symptoms: The primary symptom of actinic keratosis is the presence of rough, dry, or scaly patches on the skin. These patches can vary in color, ranging from flesh-toned to reddish-brown. They are commonly found on areas frequently exposed to the sun, such as the face, scalp, ears, neck, forearms, and hands. AK patches may itch, burn, or feel tender, especially when exposed to sunlight or touched. Over time, they can become thicker, harder, or develop a wart-like texture. Changes in color and size are also possible.









Causes: Actinic keratosis is primarily caused by excessive and prolonged exposure to ultraviolet (UV) radiation from the sun. Sunlight contains UV rays that can damage the DNA in skin cells. Individuals with fair skin, light-colored hair, and light-colored eyes are more susceptible to AK, as their skin provides less natural protection against UV radiation. Other risk factors include a history of frequent sunburns, living in sunny climates, working outdoors, and using tanning beds.

Treatment: Treating actinic keratosis is crucial to prevent its progression into skin cancer. The choice of treatment depends on factors such as the number and severity of lesions, location, and individual patient characteristics. Common treatment options include:

  1. Topical Medications: Prescription creams or gels containing 5-fluorouracil (5-FU), imiquimod, diclofenac, or ingenol mebutate are applied directly to the affected skin. These medications work to destroy AK cells over a period of weeks or months.

  2. Cryotherapy: Liquid nitrogen is used to freeze and destroy AK lesions. The freezing process causes the affected skin to blister and eventually slough off.

  3. Curettage and Electrodesiccation: The AK lesion is scraped off using a curette, and an electric needle is used to destroy any remaining abnormal cells.

  4. Photodynamic Therapy (PDT): A photosensitizing agent is applied to the AK lesions, followed by exposure to a specific wavelength of light. The light activates the agent, destroying the AK cells.

  5. Laser Therapy: Certain lasers can selectively target and destroy AK lesions.

  6. Surgical Excision: Large or thick AK lesions may require surgical removal, allowing for a more accurate examination of the tissue.

Prevention: Preventing actinic keratosis is crucial in reducing the risk of developing this condition. Protecting the skin from excessive sun exposure is key. This includes:

  • Applying broad-spectrum sunscreen with an SPF of 30 or higher, even on cloudy days.

  • Wearing protective clothing, such as wide-brimmed hats, long-sleeved shirts, and sunglasses.

  • Seeking shade and avoiding the sun during peak hours (10 am to 4 pm).

  • Avoiding tanning beds and artificial tanning methods.

  • Regularly examining the skin and promptly reporting any changes or suspicious lesions to a healthcare professional.

Regular skin checks by a Dermatologist In Dubai are recommended, especially for individuals at higher risk or with a history of actinic keratosis.

In conclusion, actinic keratosis is a common skin condition resulting from prolonged sun exposure. Recognizing its symptoms, seeking appropriate treatment, and adopting preventive measures are crucial steps in managing this condition and reducing the risk of skin cancer. By protecting the skin from excessive sun exposure and being proactive about skin health, individuals can promote their overall well-being and maintain healthy skin.

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